Cellular and Molecular Mechanisms of Drugs of Abuse and Neurotoxicity Cocaine, GHB, and Substituted Amphetamines

Name: Cellular and Molecular Mechanisms of Drugs of Abuse and Neurotoxicity Cocaine, GHB, and Substituted Amphetamines
Price: 54.0 USD
Availability: InStock
ISBN: 9781573316293

ISBN-10: 1573316296

ISBN-13: 9781573316293

Edition: 2006

Authors: Sayed Ali, Francesce Fornai, Yossef Itzhak, Sayed Ali

List price: $183.00

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Description:

Methamphetamine, MDMA, cocaine, PMA, GHB, and various solvents are the most widely abused drugs in Europe, the United States, Central America, South America, and Asia; and their use has dramatically increased over the last two decades. These drugs of abuse are known to cause neurotoxicity in several species, including not only rodents, dogs, and nonhuman primates, but also humans. The precise neurochemical mechanisms underlying this drug-induced neurotoxicity remain unclear. This volume explores this question, specifically addressing the following aspects: (1) the role of genomics and proteomics in drug-induced neurotoxicity, (2) drugs of abuse and medication development, (3) molecular…

Book details

List price: $183.00
Copyright year: 2006
Publisher: John Wiley & Sons, Incorporated
Publication date: 8/23/2006
Binding: Paperback
Pages: 552
Size: 6.00" wide x 9.00" long x 1.00" tall
Weight: 2.398
Language: English




Genes and Drug of Abuse



Opposite Regulation of Cocaine-Induced Intracellular Signaling and Gene Expression by Dopamine D1 and D3 Receptors




Serial Analysis of Gene Expression (SAGE) in the Rat Striatum following Methamphetamine Administration




Dopamine Quinones Activate Microglia and Induce a Neurotoxic Gene Expression Profile: Relationship to Methamphetamine-Induced Nerve Ending Damage




Gene expression in the brain from Fluoxetine-injected mouse by using DNA microarray




Short-term effects of adolescent methylphenidate exposure on brain striatal gene.expression and sexual/endocrine parameters in male rats




Effects of L-carnitine Pre-treatment in Methamphetamine and 3-nitropropionic acid-induced neurotoxicity




Convergent Roles of Alpha-Synuclein, DA Metabolism and the Ubiquitin-Proteasome System in Nigrostriatal Toxicity




Association Study of the Dihydropyrimidinase-related Protein 2 Gene and Methamphetamine Psychosis




Repeated methamphetamine administration alters expression of the NMDA receptor channel I?2 subunit and kinesins in the mouse brain




Genes and Gene Expression in the Brain of Human Alcoholics




Association Study of the Tumor Necrosis Factor-I? Gene and Its 1A Receptor Gene with Methampetamine Dependence




Methamphetamine Neurotoxicity



Longitudinal Clinical course following Pharmacological Treatment of Methamphetamine Psychosis which persists after long-term Abstinence




Distinct Mechanisms Mediating Methamphetamine-Induced Neuronal Apoptosis and Dopamine Terminal Damage Share the Neuropeptide Substance P in the Striatum of Mice




Effects of Methamphetamine on the Cerebellar Cortex: a Preliminary Study




Age-Dependent Effects of Methamphetamine on VMAT-2




Methamphetamine, Morphine and their Combination: Acute Changes in Striatal Dopaminergic Transmission evaluated by Microdialysis in Awake Rats




In PC12 Cells Neurotoxicity Induced by Methamphetamine is Related to Proteasome Inhibition




Human Immunodeficiency Virus-1 Protein Tat and Methamphetamine Interactions




Over Expression of I?-Synuclein Following Methamphetamine: Is it Good or Bad?




Alterations in Blood-Brain Barrier Function by Morphine and Methamphetamine